J. D. Pickard, N. Akalan, V. V. Dolenc, J. J. A. Mooij's Advances and Technical Standards in Neurosurgery PDF

By J. D. Pickard, N. Akalan, V. V. Dolenc, J. J. A. Mooij

ISBN-10: 3211474161

ISBN-13: 9783211474167

This sequence, subsidized through the ecu organization of Neurosurgical Societies, has already turn into a vintage. regularly, one quantity is released consistent with yr. The Advances part provides fields of neurosurgery and comparable components within which very important fresh growth has been made. The Technical criteria part positive factors distinct descriptions of normal methods to help younger neurosurgeons of their post-graduate education. The contributions are written via skilled clinicians and are reviewed by means of all contributors of the Editorial Board.

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Czeh G, Aitken PG, Somjen GG (1993) Membrane currents in CA1 pyramidal cells during spreading depression (SD) and SD-like hypoxic depolarization. Brain Res 632:195–208 Depolarisation Phenomena in Traumatic and Ischaemic Brain Injury 41 18. Snow RW, Taylor CP, Dudek FE (1983) Electrophysiological and optical changes in slices of rat hippocampus during spreading depression. J Neurophysiol 50:561–572 19. Hasegawa Y, Latour LL, Formato JE, Sotak CH, Fisher M (1995) Spreading waves of a reduced di¤usion coe‰cient of water in normal and ischemic rat brain.

The individual appears to be completely alert and can commu- 30 A. J. Strong and R. Dardis nicate, but enquires frequently about present events. Most episodes last around eight hours but can last for 24 hours and indeed an episode lasting seven days has been described. The onset of memory loss may occur during an emotional stimulus or physical exertion. A history of migraine is recognised in up to 25% of TGA patients. Cerebral blood flow studies using the 133-xenon inhalation method in TGA patients suggest temporary regional hypoperfusion [114].

Characterised noninvasively the transient changes in oxidised and reduced haemoglobin (HbO, Hb) that accompany CSD in the rat brain [136]. A transient increase in HbO was accompanied by a reduction in Hb, a combination suggesting hyperaemia (Fig. 10, upper). There are clinical NIRS data from this department in one patient with TBI (hitherto unpublished: Fig. 10 lower) and from the Berlin group in two with ischaemic stroke (Fig. 11), in which the HbO and Hb transients closely resembled those seen with CSD in the laboratory.

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Advances and Technical Standards in Neurosurgery by J. D. Pickard, N. Akalan, V. V. Dolenc, J. J. A. Mooij

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